Impaired cerebrovascular reactivity in obstructive sleep apnea: A case-control study

Obstructive sleep apnea (OSA) is an independent risk factor for stroke. Little is known about the cerebrovascular hemodynamic changes during apnea. Hypercapnia occurs in apneas and hypopneas, and a reduced cerebral vasodilatory response to CO2 could compromise the cerebral blood flow (CBF). Therefore, we aimed to evaluate whether the apnea−hypopnea index (AHI) affected the cerebrovascular response to CO2.

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