Mold exposure is considered a risk factor for allergic diseases such as AD, although with some debate.1 The inconsistency of the results obtained in regard to this association may be due in part to differences in the dosage, time, and duration of the exposure. Skin barrier dysfunction and immune dysregulation have recently been highlighted as key factors in the development of AD. The transepidermal water loss (TEWL) level reflects skin barrier integrity, the loss of which is one of the most important features of AD.
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