Abstract
Linburg-Comstock syndrome is characterized by an inability to flex the interphalangeal joint of the thumb without simultaneous flexion of the distal interphalangeal joint of the index finger due to hereditary interconnections between the flexor pollicis longus (FPL) and the index flexor digitorum profundus (iFDP) resulting in discomfort and symptoms of flexor tenosynovitis. In addition to this anatomic anomaly, our clinical findings suggest that the interconnection can also result secondarily as a consequence of tenosynovial hyperplasia producing adhesions from any cause including previous surgery or trauma using the example of forearm laceration with dissection of the two tendons.
Level of Evidence: Level V, diagnostic study.
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